“It has been shown that the feeding of desiccated placenta to women during the first eleven days after parturition causes an increase in the protein and lactose percent of the milk… All the mothers were receiving the same diet, and to the second set 0.6mg of desiccated placenta was fed three times a day throughout the period. Certain definite differences in the progress of growth of the two sets of infants are to be observed. It is evident that the recovery from the postnatal decline in weight is hastened by the consumption of milk produced under the influence of maternally ingested placenta.” - McNeile, Lyle G. 1918. The American journal of obstetrics and diseases of women and children, 77. W.A. Townsend & Adams, original press: University of Michigan
"An attempt was made to increase milk secretion in mothers by administration of dried placenta per os. Of 210 controlled cases only 29 (13.8%) gave negative results; 181 women (86.2%) reacted positively to the treatment, 117 (55.7%) with good and 64 (30.5%) with very good results. It could be shown by similar experiments with a beef preparation that the effective substance in placenta is not protein. Nor does the lyofilised placenta act as a biogenic stimulator so that the good results of placenta administration cannot be explained as a form of tissue therapy per os. The question of a hormonal influence remains open. So far it could be shown that progesterone is probably not active in increasing lactation after administration of dried placenta." - Placenta as Lactagagon: Soykova-Pachnerova E, et. al.(1954). Gynaecologia 138(6):617-627
"In rats that were allowed to eat the placentae after parturition concentrations of serum prolactin were elevated on Day 1 but concentrations of serum progesterone were depressed on Days 6 and 8 post partum when compared to those of rats prevented from eating the placentae. In rats treated with PMSG to induce superovulation serum prolactin and progesterone values were significantly (P < 0.05) elevated on Days 3 and 5 respectively, after being fed 2 g rat placenta/day for 2 days. However, feeding each rat 4 g placenta/day significantly (P < 0.02) lowered serum progesterone on Day 5. Oestrogen injections or bovine or human placenta in the diet had no effect. The organic phase of a petroleum ether extract of rat placenta (2 g-equivalents/day) lowered peripheral concentrations of progesterone on Day 5, but other extracts were ineffective. We conclude that the rat placenta contains orally-active substance(s) which modify blood levels of pituitary and ovarian hormones." - Effects of placentophagy on serum prolactin and progesterone concentrations in rats after parturition or superovulation. Blank MS, Friesen HG.: J Reprod Fertil. 1980 Nov;60(2):273-8.
“Powdered Placenta Hominis was used for 57 cases of insufficient lactation. Within 4 days, 48 women had markedly increased milk production, with the remainder following suit over the next three days.” - Bensky/Gamble. 1997. Materia Medica, Eastland Press, 54
"Ingestion of placenta or amniotic fluid produces a dramatic enhancement of centrally mediated opioid antinociception in the rat. The present experiments investigated the role of each opioid receptor type (A, y, n) in the antinociception-modulating effects of Placental Opioid-Enhancing Factor (POEF—presumably the active substance). Antinociception was measured on a 52 jC hotplate in adult, female rats after they ingested placenta or control substance (1.0 g) and after they received an intracerebroventricular injection of a y-specific ([D-Pen2,D-Pen5]enkephalin (DPDPE); 0, 30, 50, 62, or 70 nmol), A-specific ([D-Ala2,N-MePhe4,Gly5-ol]enkephalin (DAMGO); 0, 0.21, 0.29, or 0.39 nmol), or n-specific (U-62066; spiradoline; 0, 100, 150, or 200 nmol) opioid receptor agonist. The results showed that ingestion of placenta potentiated y- and n-opioid antinociception, but attenuated A-opioid antinociception. This finding of POEF action as both opioid receptor-specific and complex provides an important basis for understanding the intrinsic pain-suppression mechanisms that are activated during parturition and modified by placentophagia, and important information for the possible use of POEF as an adjunct to opioids in pain management." -Placenta for Pain Relief: Placenta ingestion by rats enhances y- and n-opioid antinociception, but suppresses A-opioid antinociceptio Jean M. DiPirro*, Mark B. Kristal
Estrogen, Progesterone, Testosterone Contributes to mammary gland development in preparation for lactation; stabilizes postpartum mood; regulates post-birth uterine cramping; decreases depression; normalizes and stimulates libido Prolactin Promotes lactation; increases milk supply; enhances the mothering instinct Corticotropin Releasing Hormone (CRH) Low levels of CRH are implicated in postpartum depression. Regulation of CRH helps prevent depression Oxytocin Decreases pain and increases bonding in mother and infant; counteracts the production of stress hormones such as Cortisol; greatly reduces postpartum bleeding; enhances the breastfeeding let-down reflex Placental Opioid-Enhancing Factor (POEF) Stimulates the production of your body’s natural opioids, including endorphins; reduces pain, produces feelings of well-being Thyroid Stimulating Hormone Regulates the thyroid gland; boosts energy and supports recovery from stressful events Cortisone Reduces inflammation and swelling; promotes healing Interferon Triggers the protective defenses of the immune system to fight infection Prostaglandins Regulates contractions in the uterus after birth; helps uterus return to its pre-pregnancy size. Anti-inflammatory effects Iron Iron combats anemia, increases energy; reduces fatigue and depression Hemoglobin Oxygen-carrying molecule which provides a boost in energy Urokinase Inhibiting Factor and Factor XIII Stops bleeding and enhances wound healing Immunoglobulin G (IgG) Antibody molecules which support the immune system Human Placental Lactogen (hPL) This hormone has lactogenic and growth-promoting properties; promotes mammary gland growth in preparation for lactation in the mother. It also regulates maternal glucose, protein, and fat levels
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